We publish our audit record because accuracy claims should be checkable. Every entry on this site runs through an automated fact-audit pipeline (accuracy audit → source-grounded repair → visual QA); this page is generated from that pipeline's own report, not written by hand.
Last full accuracy audit: 2026-09-07 · 20 entries checked · 7 flagged · 13 issues confirmed · 7 corrected · 6 still open
Chimera (genetics) — The claim that '95% of marmoset fraternal twins trade blood through chorionic fusions' is inaccurate; the widely accepted figure is that nearly 100% of marmoset fraternal twins exhibit chimerism due to placental fusion.
Chimera (genetics) — The claim that 'Male yellow crazy ants are obligate chimeras, the first known such case in animals' is misleading because other examples of obligate chimeras in animals exist, such as in certain marine sponges.
Human chimera — The 1953 case of a woman with two blood types from her twin brother is misdated; the first documented case of natural chimerism involving blood types was reported in 1953 by Dr. Ivor Dunsford and colleagues, but the woman was not found to have cells from her t
Klinefelter syndrome — The claim 'Approximately 50% of males with Klinefelter syndrome can produce sperm' is factually incorrect; the vast majority are infertile, with only a small minority (often less than 10%, typically in mosaic forms) possibly producing sperm.
Klinefelter syndrome — The claim 'Women at 40 years have a four-times-higher risk of a child with Klinefelter syndrome than women aged 24 years' is imprecise and misleading; while older maternal age is a risk factor, a specific 4x multiplier for age 40 vs. 24 is not a standard, wide
Mosaic variegated aneuploidy syndrome — The first mention of MVA was made by Rudd and colleagues in 1983 is incorrect; the earliest known description is often attributed to Warburton and colleagues in 1991, as Rudd described mosaic trisomy 8, not MVA.
Mosaic variegated aneuploidy syndrome — The disorder is caused by defects in genes responsible for the spindle checkpoint is partially incorrect; BUB1 mutations are not a confirmed cause of MVA, as established causes are mutations in BUB1B, BUB3, CEP57, and TRIP13.
Müllerian anomalies — The entry states that genetic causes include mutations in WNT3, but the correct gene for MRKH syndrome is WNT4, not WNT3.
Müllerian anomalies — The entry states 'deletion of 17q12, resulting in loss of the HNFB and LHX1 genes', but the correct gene name is HNF1B, not HNFB.
RCCX — The entry claims that with two or more modules, only one copy of each functional gene remains except for C4, but in bimodular or trimodular structures, multiple functional copies of CYP21 and TNX can exist, and not all C4 copies are necessarily active.
RCCX — The entry states each C4 gene contains 41 exons, but this is only true for the long form; the short form has 39 exons, so the statement is misleading without specifying the variant.
Ring chromosome 22 — The claim that ring chromosome 22 was first identified in 1970 is incorrect; it was first described in 1968 by Lejeune et al.
Ring chromosome 22 — The claim that Neurofibromatosis type II occurs in a significant minority of ring 22 cases is misleading; NF2 is associated with deletions of the NF2 gene on chromosome 22, not a typical feature of ring chromosome 22 syndrome.
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